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CJC-1295 (specifically the DAC-conjugated form at CJC 1295 NO DAC) extends the half-life of endogenous growth hormone-releasing hormone (GHRH) by binding to serum albumin, which preserves the natural pulsatile secretion pattern the pituitary evolved to respond to

Careful monitoring of side effects during IC clinical trials is crucial

Our observations and consequent hypothesis now provide a vehicle to explore this proposal

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